By Moore R. L.
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Extra info for Concerning Paths That Do Not Separate a Given Continuous Curve
2006; Lackland & Egan, 2007). Pathogenesis is also not known. Because BP equals cardiac output (CO) × total peripheral vascular resistance (TPR), pathogenic mechanisms must involve increased CO, increased TPR, or both. Many theories have been proposed to explain this equation; the microcirculation theory is the most attractive among them. In accordance with this theory the primary defect involves small resistance vessels, leading to increased TPR and sustained 22 Genetics and Pathophysiology of Essential Hypertension elevated BP.
2001). , 1994). A perennial problem in understanding the interaction between kidney and hypertension is the poor correlation between hypertension, and vascular and glomerular lesion. This is in part due to these lesions being present to a greater or lesser degree in the normotensive, aging kidney, with racial differences in severity further confounding the problem. Recent experimental and clinical data suggest that functional impairment and vasoconstriction in afferent arterioles (renal autoregulation) precede morphologic lesions.
1993). Recently, ample interest has been devoted to the relationship between arterial stiffness and cardiovascular disease. , 1997). Arterial stiffness may be measured using a variety of different techniques, mainly ultrasound based. Applanation tonometry pulse wave velocity is the most commonly used parameter in detecting central arterial stiffness (Nichols, 2005). , 2001). , 1994). A perennial problem in understanding the interaction between kidney and hypertension is the poor correlation between hypertension, and vascular and glomerular lesion.
Concerning Paths That Do Not Separate a Given Continuous Curve by Moore R. L.