By Icon Health Publications
This can be a 3-in-1 reference e-book. It supplies a whole scientific dictionary protecting thousands of phrases and expressions with regards to cirrhosis. It additionally supplies wide lists of bibliographic citations. eventually, it offers info to clients on the best way to replace their wisdom utilizing quite a few web assets. The ebook is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to familiarize yourself with examine devoted to cirrhosis. in case your time is effective, this e-book is for you. First, you won't waste time looking the net whereas lacking loads of suitable info. moment, the ebook additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing enormous quantities of websites.
Read or Download Cirrhosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References PDF
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Extra info for Cirrhosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
We have shown that bile acids inhibit hepatic glucagoninduced cAMP synthesis at physiologic concentrations. The effect was at the level of receptor-Gs coupling, most likely through phosphorylation, and was mediated by a calcium-dependent PKC. We have reported that hepatic glucagon-mediated cAMP production was attenuated in cholestasis in hamster induced by ligation of the common bile duct (BDL). Bile acids were either without or with reduced effects after BDL suggesting that the site of cAMP synthesis cascade altered in cholestasis is the same as that altered by bile acids.
The Specific Aims are: 1. To generate recombinant baculoviruses that contain all or part of the HCV cDNA under the control of mammalian promoters. 2. To test the ability of HCV-recombinant baculoviruses to express HCV gene products and replicate HCV in human cells of hepatic origin. 3. To use recombinant HCV baculovirus to evaluate the direct effect of interferon treatment on evolution of quasispecies during HCV replication in cells of hepatic origin. ; Director, Div. Gastroenterology; Internal Medicine; University of California Davis Sponsored Programs, 118 Everson Hall Davis, Ca 95616 Timing: Fiscal Year 2001; Project Start 01-SEP-1988; Project End 31-MAR-2006 Summary: This proposal is a continuation of our studies which attempt to elucidate the molecular bases of cirrhosis in alcoholic liver disease.
The research will examine acute alcohol impairment of cognitive functions in young non-dependent drinkers. The project combines measures of alcohol effects on cognitive inhibitory processes with traditional abuse liability indices based on subjective rewarding effects of the drug and its ability to reinforce self-administration. , suppression of an avoidance "stop" mechanism). Multiple strategies will test the role of acute cognitive impairment in the abuse liability of alcohol. A drug-reinforcement model will test the degree to which preload alcohol doses "prime" subsequent drug self-administration by impairing inhibitory control processes that regulate behavior.
Cirrhosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by Icon Health Publications